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Kindling hypothesis
From MEpedia, a crowd-sourced encyclopedia of ME and CFS science and history
 | This article is a stub. |
The Kindling hypothesis attempts to explain the hypersensitivities to pain, sensory input, and the immune system responses found in patients with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.[1] Kindling "occurs when an organism is exposed repeatedly to an initially sub-threshold stimulus resulting in hypersensitivity and spontaneous seizure-like activity."
Theory[edit | edit source]
Evidence[edit | edit source]
Treatment[edit | edit source]
See also[edit | edit source]
- Medical hypotheses (category)
- Leonard Jason
- Sensitivies
- Pain
Learn more[edit | edit source]
- An Etiological Model for Myalgic Encephalomyelitis/Chronic Fatigue Syndrome[1] (Full text)
References[edit | edit source]
- ↑ Jump up to: 1.0 1.1 Jason, Leonard A.; Sorenson, Matthew; Porter, Nicole; Belkairous, Natalie (March 1, 2011). "An Etiological Model for Myalgic Encephalomyelitis/Chronic Fatigue Syndrome". Neuroscience and medicine. 2 (1): 14–27. doi:10.4236/nm.2011.21003. ISSN 2158-2912. PMC 3166239. PMID 21892413.
