Vincent Lombardi

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Source:Nevada Center for Biomedical Research

Vincent C. Lombardi, Ph.D., was the Director of Research at the Whittemore Peterson Institute. He has been researching myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) since 1992 in collabration with Dr. Daniel Peterson and Dr. Kenny De Meirleir. After completing his academic studies, he joined the research team of the Whittemore Peterson Institute, rising to director in 2011.[1]

In September of 2018, Dr. Lombardi joined the University of Nevada, Reno research community as an Associate Professor Research in the Department of Microbiology.[2] Dr. Lombardi continues to work closely with WPI's expert physician, Dr. Kenny De Meirleir. Dr. De Meirleir and Dr. Lombardi have worked closely together for years in an effort to solve ME/CFS and similar infection-associated neuroimmune diseases.[3]

Notable studies[edit | edit source]

  • 2016, Humoral Immunity Profiling of Subjects with Myalgic Encephalomyelitis Using a Random Peptide Microarray Differentiates Cases from Controls with High Specificity and Sensitivity[4] - (Full text)
  • 2016, Genome-wide association analysis identifies genetic variations in subjects with myalgic encephalomyelitis/chronic fatigue syndrome[5] - (Full text)
  • 2015, Cytokine expression provides clues to the pathophysiology of Gulf War illness and myalgic encephalomyelitis[6] - (Full text)
  • 2014, Plasmacytoid dendritic cells of the gut: Relevance to immunity and pathology[7] - (Full text)
  • 2013, Plasmacytoid dendritic cells in the duodenum of individuals diagnosed with myalgic encephalomyelitis are uniquely immunoreactive to antibodies to human endogenous retroviral proteins[8] - (Full text)

Talks and interviews[edit | edit source]

Online presence[edit | edit source]

See also[edit | edit source]

Learn more[edit | edit source]

References[edit | edit source]

  1. ↑ NVCBR. "Vincent C. Lombardi, PhD". Whittemore Peterson Institute. Retrieved July 15, 2019.
  2. ↑ "University Directory". apps.unr.edu. Retrieved June 10, 2019.
  3. ↑ NVCBR. "Research". Whittemore Peterson Institute. Retrieved June 10, 2019.
  4. ↑ Singh, Sahajpreet; Stafford, Phillip; Schlauch, Karen A.; Tillett, Richard R.; Gollery, Martin; Johnston, Stephen Albert; Khaiboullina, Svetlana F.; De Meirleir, Kenny L.; Rawat, Shanti; Mijatovic, Tatjana; Subramanian, Krishnamurthy; Palotás, András; Lombardi, Vincent C. (2016), "Humoral Immunity Profiling of Subjects with Myalgic Encephalomyelitis Using a Random Peptide Microarray Differentiates Cases from Controls with High Specificity and Sensitivity", Molecular Neurobiology, doi:10.1007/s12035-016-0334-0
  5. ↑ Schlauch, Karen A.; Khaiboullina, Svetlana F.; De Meirleir, Kenny L.; Rawat, Shanti; Petereit, J; Rizvanov, Albert A; Blatt, Nataliya; Mijatovic, Tatjana; Kulick, D; Palotás, András; Lombardi, Vincent C. (2016), "Genome-wide association analysis identifies genetic variations in subjects with myalgic encephalomyelitis/chronic fatigue syndrome", Translational Psychiatry, 6 (2): e730, doi:10.1038/tp.2015.208
  6. ↑ Khaiboullina, Svetlana F.; DeMeirleir, Kenny L.; Rawat, Shanti; Berk, Grady S.; Gaynor-Berk, Rory S.; Mijatovic, Tatjana; Blatt, Natalia; Rizvanov, Albert A.; Young, Sheila G. (March 2015). "Cytokine expression provides clues to the pathophysiology of Gulf War illness and myalgic encephalomyelitis". Cytokine. 72 (1): 1–8. doi:10.1016/j.cyto.2014.11.019. ISSN 1096-0023. PMC 4410698. PMID 25514671.
  7. ↑ Lombardi, Vincent C.; Khaiboullina, Svetlana F. (July 1, 2014). "Plasmacytoid dendritic cells of the gut: Relevance to immunity and pathology". Clinical Immunology. 153 (1): 165–177. doi:10.1016/j.clim.2014.04.007. ISSN 1521-6616.
  8. ↑ De Meirleir, Kenny L.; Khaiboullina, Svetlana F.; Frémont, Marc; Hulstaert, Jan; Rizvanov, Albert A.; Palotás, András; Lombardi, Vincent C. (March 2013). "Plasmacytoid dendritic cells in the duodenum of individuals diagnosed with myalgic encephalomyelitis are uniquely immunoreactive to antibodies to human endogenous retroviral proteins". In Vivo (Athens, Greece). 27 (2): 177–187. ISSN 1791-7549. PMC 3776582. PMID 23422476.

myalgic encephalomyelitis (M.E.) - A disease often marked by neurological symptoms, but fatigue is sometimes a symptom as well. Some diagnostic criteria distinguish it from chronic fatigue syndrome, while other diagnostic criteria consider it to be a synonym for chronic fatigue syndrome. A defining characteristic of ME is post-exertional malaise (PEM), or post-exertional neuroimmune exhaustion (PENE), which is a notable exacerbation of symptoms brought on by small exertions. PEM can last for days or weeks. Symptoms can include cognitive impairments, muscle pain (myalgia), trouble remaining upright (orthostatic intolerance), sleep abnormalities, and gastro-intestinal impairments, among others. An estimated 25% of those suffering from ME are housebound or bedbound. The World Health Organization (WHO) classifies ME as a neurological disease.

genome an organism's complete set of DNA, including all of its genes

antibodies Antibody/immunoglobulin refers to any of a large number of specific proteins produced by B cells that act against an antigen in an immune response.

antibodies Antibody/immunoglobulin refers to any of a large number of specific proteins produced by B cells that act against an antigen in an immune response.

endogenous Growing or originating from within an organism.

cytokine any class of immunoregulatory proteins secreted by cells, especially immune cells. Cytokines are small proteins important in cell signaling that modulate the immune system. (Learn more: me-pedia.org)

The information provided at this site is not intended to diagnose or treat any illness.
From MEpedia, a crowd-sourced encyclopedia of ME and CFS science and history.

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