ADAM metallopeptidase with thrombospondin type 1 motif 19: Difference between revisions

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== Notable studies ==
== Notable studies ==
* 2019, Genetic Predisposition for Immune System, Hormone, and Metabolic Dysfunction in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Pilot Study<ref name="Nathanson2019">{{Cite journal | last = Nathanson | first = Lubov | authorlink = Lubov Nathanson | authorlink2 = Travis Craddock | last2 = Craddock | first2 = Travis J. A. | last3 = Klimas | first3 = Nancy G. | author-link3=Nancy Klimas  | authorlink4 = Kristina Gemayel | last4 = Gemayel | first4 = Kristina | last5 = Del Alamo | first5 = Ana  | authorlink5 = Ana Del Alamo | authorlink6 = Kelly Hilton | last6 = Hilton | first6 = Kelly | last7 = Jaundoo | first7 = Rajeev | authorlink7 = Rajeev Jaundoo | last8 = Perez | first8 = Melanie | authorlink8 = Melanie Perez | date = 2019 | title = Genetic Predisposition for Immune System, Hormone, and Metabolic Dysfunction in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Pilot Study | url = https://www.frontiersin.org/articles/10.3389/fped.2019.00206/full|journal=Frontiers in Pediatrics|language=English|volume=7|issue= | pages = 206|doi=10.3389/fped.2019.00206|issn=2296-2360}}</ref> [https://doi.org/10.3389/fped.2019.00206 (Full text)]
* 2019, Genetic Predisposition for Immune System, Hormone, and Metabolic Dysfunction in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Pilot Study<ref name="Nathanson2019">{{Cite journal | last = Nathanson | first = Lubov | authorlink = Lubov Nathanson | authorlink2 = Travis Craddock | last2 = Craddock | first2 = Travis J.A. |last3 = Klimas | first3 = Nancy G. | author-link3=Nancy Klimas  | authorlink4 = Kristina Gemayel | last4 = Gemayel | first4 = Kristina | last5 = Del Alamo | first5 = Ana  | authorlink5 = Ana Del Alamo | authorlink6 = Kelly Hilton | last6 = Hilton | first6 = Kelly | last7 = Jaundoo | first7 = Rajeev | authorlink7 = Rajeev Jaundoo | last8 = Perez | first8 = Melanie | authorlink8 = Melanie Perez | date = 2019 | title = Genetic Predisposition for Immune System, Hormone, and Metabolic Dysfunction in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Pilot Study | url = https://www.frontiersin.org/articles/10.3389/fped.2019.00206/full|journal=Frontiers in Pediatrics|language=English|volume=7|issue= | pages = 206|doi=10.3389/fped.2019.00206|issn=2296-2360}}</ref> [https://doi.org/10.3389/fped.2019.00206 (Full text)]


==See also==
==See also==

Revision as of 11:08, November 30, 2022

ADAMTS19 or ADAM metallopeptidase with thrombospondin type 1 motif 19 or ADAM metallopeptidase domain 19 or A Disintegrin-Like And Metalloprotease (Reprolysin Type) With Thrombospondin Type 1 Motif, 19 is a protein-encoding gene.[1]

Function[edit | edit source]

ADAMTS19 is associated with discrete subaortic stenosis and subvalvular aortic stenosis.[1]

ME/CFS[edit | edit source]

The first publication from the long term ME/CFS Gene Study, which used the Fukuda criteria for chronic fatigue syndrome only, looked for the SNPs that were more common in CFS patients than in healthy people, and which were most likely to be harmful. ADAMTS19 was one of the top ten genes most likely to be harmful that more common in CFS patients, occurring in over 70% of patients.[2]

Notable studies[edit | edit source]

  • 2019, Genetic Predisposition for Immune System, Hormone, and Metabolic Dysfunction in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Pilot Study[2] (Full text)

See also[edit | edit source]

Learn more[edit | edit source]

References[edit | edit source]