Editing Michael Maes
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==Books== | ==Books== | ||
*2011, ''[[Nooit Meer Moe]]'' (English translation: 'Tired No More, CFS Unmasked') <ref name="Michael Maes, 2011">{{Cite book | author = Michael Maes| title = Nooit-Mer-Moe | trans-title = Never Tired| language = nl| date = Apr 2011| publisher = Zorro Publisher| isbn = 9789461680013 | url = https://www.bol.com/nl/p/nooit-meer-moe/1001004011100533/ }}</ref> | *2011, ''[[Nooit Meer Moe]]'' (English translation: 'Tired No More, CFS Unmasked') <ref name="Michael Maes, 2011">{{Cite book | author = Michael Maes| title = Nooit-Mer-Moe | trans-title = Never Tired| language = nl| date = Apr 2011| publisher = Zorro Publisher| isbn = 9789461680013 | url = https://www.bol.com/nl/p/nooit-meer-moe/1001004011100533/ }}</ref> | ||
==Notable studies== | ==Notable studies== | ||
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*2015, Central pathways causing fatigue in neuro-inflammatory and autoimmune illnesses<ref name="Morris, 2015">{{Cite journal | last1 = Morris | first1 = Gerwyn | authorlink1 = Gerwyn Morris | last2 = Berk | first2 = Michael | authorlink2 = Michael Berk | last3 = Walder | first3 = K | authorlink3 = | last4 = Maes | first4 = Michael | authorlink4 = Michael Maes | title = Central pathways causing fatigue in neuro-inflammatory and autoimmune illnesses.| journal = BMC Medicine | volume = 13 | issue = | pages = 28 | date = 2015 | url = https://bmcmedicine.biomedcentral.com/track/pdf/10.1186/s12916-014-0259-2 | *2015, Central pathways causing fatigue in neuro-inflammatory and autoimmune illnesses<ref name="Morris, 2015">{{Cite journal | last1 = Morris | first1 = Gerwyn | authorlink1 = Gerwyn Morris | last2 = Berk | first2 = Michael | authorlink2 = Michael Berk | last3 = Walder | first3 = K | authorlink3 = | last4 = Maes | first4 = Michael | authorlink4 = Michael Maes | title = Central pathways causing fatigue in neuro-inflammatory and autoimmune illnesses.| journal = BMC Medicine | volume = 13 | issue = | pages = 28 | date = 2015 | url = https://bmcmedicine.biomedcentral.com/track/pdf/10.1186/s12916-014-0259-2 | ||
| doi = 10.1186/s12916-014-0259-2}}</ref> - [https://bmcmedicine.biomedcentral.com/track/pdf/10.1186/s12916-014-0259-2 (Full Text)] | | doi = 10.1186/s12916-014-0259-2}}</ref> - [https://bmcmedicine.biomedcentral.com/track/pdf/10.1186/s12916-014-0259-2 (Full Text)] | ||
*2016, Nitrosative Stress, Hypernitrosylation, and Autoimmune Responses to Nitrosylated Proteins: New Pathways in Neuroprogressive Disorders Including Depression and Chronic Fatigue Syndrome<ref name="Morris, 23Jun2016">{{Cite journal | last1 = Morris | first1 = Gerwyn | authorlink1 = Gerwyn Morris | last2 = Berk | first2 = Michael | authorlink2 = Michael Berk | last3 = Klein | first3 = H. | authorlink3 = | last4 = Walder | first4 = K. | authorlink4 = | last5 = Galacki | first5 = Piotr | authorlink5 = | last6 = Maes | first6 = Michael | authorlink6 = Michael Maes | title = Nitrosative Stress, Hypernitrosylation, and Autoimmune Responses to Nitrosylated Proteins: New Pathways in Neuroprogressive Disorders Including Depression and Chronic Fatigue Syndrome| journal = Molecular Neurobiology | volume = 54 | issue = 6 | pages = 4271-4291 | date = Jun 23, 2016 | url = http://link.springer.com/article/10.1007/s12035-016-9975-2 | doi = 10.1007/s12035-016-9975-2}}</ref> - [http://link.springer.com/article/10.1007/s12035-016-9975-2 (Abstract)] | *2016, Nitrosative Stress, Hypernitrosylation, and Autoimmune Responses to Nitrosylated Proteins: New Pathways in Neuroprogressive Disorders Including Depression and Chronic Fatigue Syndrome<ref name="Morris, 23Jun2016">{{Cite journal | last1 = Morris | first1 = Gerwyn | authorlink1 = Gerwyn Morris | last2 = Berk | first2 = Michael | authorlink2 = Michael Berk | last3 = Klein | first3 = H. | authorlink3 = | last4 = Walder | first4 = K. | authorlink4 = | last5 = Galacki | first5 = Piotr | authorlink5 = | last6 = Maes | first6 = Michael | authorlink6 = Michael Maes | title = Nitrosative Stress, Hypernitrosylation, and Autoimmune Responses to Nitrosylated Proteins: New Pathways in Neuroprogressive Disorders Including Depression and Chronic Fatigue Syndrome| journal = Molecular Neurobiology | volume = 54 | issue = 6 | pages = 4271-4291 | date = Jun 23, 2016 | url = http://link.springer.com/article/10.1007/s12035-016-9975-2 | doi = 10.1007/s12035-016-9975-2}}</ref> - [http://link.springer.com/article/10.1007/s12035-016-9975-2 (Abstract)] | ||
*2016, The role of microbiota and intestinal permeability in the pathophysiology of autoimmune and neuroimmune processes with an emphasis on Inflammatory Bowel Disease, Type 1 Diabetes and Chronic Fatigue Syndrome<ref name="Morris, 2016">{{Cite journal | last1 = Morris | first1 = Gerwyn | authorlink1 = Gerwyn Morris | last2 = Berk | first2 = Michael | authorlink2 = Michael Berk | last3 = Carvalho | first3 = A.F. | authorlink3 = | last4 = Caso | first4 = J.R. | authorlink4 = | last5 = Sanz | first5 = Y. | authorlink5 = | last6 = Maes | first6 = Michael | authorlink6 = Michael Maes | title = The role of microbiota and intestinal permeability in the pathophysiology of autoimmune and neuroimmune processes with an emphasis on Inflammatory Bowel Disease Type 1 Diabetes and Chronic Fatigue Syndrome.| journal = Current Pharmaceutical Design | volume = 22 | issue = 40 | pages = 6058-6075 | date = 2016 | url = https://www.ncbi.nlm.nih.gov/pubmed/27634186 | pmid = 27634186}}</ref> - [https://www.ncbi.nlm.nih.gov/pubmed/27634186 (Abstract)] | *2016, The role of microbiota and intestinal permeability in the pathophysiology of autoimmune and neuroimmune processes with an emphasis on Inflammatory Bowel Disease, Type 1 Diabetes and Chronic Fatigue Syndrome<ref name="Morris, 2016">{{Cite journal | last1 = Morris | first1 = Gerwyn | authorlink1 = Gerwyn Morris | last2 = Berk | first2 = Michael | authorlink2 = Michael Berk | last3 = Carvalho | first3 = A.F. | authorlink3 = | last4 = Caso | first4 = J.R. | authorlink4 = | last5 = Sanz | first5 = Y. | authorlink5 = | last6 = Maes | first6 = Michael | authorlink6 = Michael Maes | title = The role of microbiota and intestinal permeability in the pathophysiology of autoimmune and neuroimmune processes with an emphasis on Inflammatory Bowel Disease Type 1 Diabetes and Chronic Fatigue Syndrome.| journal = Current Pharmaceutical Design | volume = 22 | issue = 40 | pages = 6058-6075 | date = 2016 | url = https://www.ncbi.nlm.nih.gov/pubmed/27634186 | pmid = 27634186}}</ref> - [https://www.ncbi.nlm.nih.gov/pubmed/27634186 (Abstract)] | ||
*2017, Mechanisms Explaining Muscle Fatigue and Muscle Pain in Patients with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): a Review of Recent Findings<ref name="Morris, 2017">{{Cite journal | last1 = Morris | first1 = Gerwyn | authorlink1 = Gerwyn Morris | last2 = Maes | first2 = Michael | authorlink2 = Michael Maes | title = Mechanisms Explaining Muscle Fatigue and Muscle Pain in Patients with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): a Review of Recent Findings| journal = Current Rheumatology Reports | volume = 19 | issue = 1 | pages = 1 | date = 2017| url = http://link.springer.com/article/10.1007/s11926-017-0628-x | pmid = | doi = 10.1007/s11926-017-0628-x}}</ref> - [http://link.springer.com/article/10.1007/s11926-017-0628-x (Abstract)] | *2017, Mechanisms Explaining Muscle Fatigue and Muscle Pain in Patients with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): a Review of Recent Findings<ref name="Morris, 2017">{{Cite journal | last1 = Morris | first1 = Gerwyn | authorlink1 = Gerwyn Morris | last2 = Maes | first2 = Michael | authorlink2 = Michael Maes | title = Mechanisms Explaining Muscle Fatigue and Muscle Pain in Patients with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS): a Review of Recent Findings| journal = Current Rheumatology Reports | volume = 19 | issue = 1 | pages = 1 | date = 2017| url = http://link.springer.com/article/10.1007/s11926-017-0628-x | pmid = | doi = 10.1007/s11926-017-0628-x}}</ref> - [http://link.springer.com/article/10.1007/s11926-017-0628-x (Abstract)] | ||
*2018, Recognizing the leaky gut as a trans-diagnostic target for neuro-immune disorders using clinical chemistry and molecular immunology assays<ref name="Simeonova2018">{{Cite journal | last = Simeonova | first = Denitsa | authorlink = | last2 = Ivanovska | first2 = Mariya | authorlink2 = | last3 = Murdjeva | first3 = Mariana | author-link3 = | last4 = Carvalho | first4 = Andre F. | authorlink4 = | last5 = Maes | first5 = Michael | authorlink5 = Michael Maes | date = 2018-11-15 | title = Recognizing the leaky gut as a trans-diagnostic target for neuro-immune disorders using clinical chemistry and molecular immunology assays|url=http://www.eurekaselect.com/167390/article|journal=Current Topics in Medicinal Chemistry|language=en|volume=18|issue=19|pages=1641-1655|doi=10.2174/1568026618666181115100610|issn=1568-0266|quote=|via=}}</ref> - [[pubmed:30430944|(Abstract)]] | *2018, Recognizing the leaky gut as a trans-diagnostic target for neuro-immune disorders using clinical chemistry and molecular immunology assays<ref name="Simeonova2018">{{Cite journal | last = Simeonova | first = Denitsa | authorlink = | last2 = Ivanovska | first2 = Mariya | authorlink2 = | last3 = Murdjeva | first3 = Mariana | author-link3 = | last4 = Carvalho | first4 = Andre F. | authorlink4 = | last5 = Maes | first5 = Michael | authorlink5 = Michael Maes | date = 2018-11-15 | title = Recognizing the leaky gut as a trans-diagnostic target for neuro-immune disorders using clinical chemistry and molecular immunology assays|url=http://www.eurekaselect.com/167390/article|journal=Current Topics in Medicinal Chemistry|language=en|volume=18|issue=19|pages=1641-1655|doi=10.2174/1568026618666181115100610|issn=1568-0266|quote=|via=}}</ref> - [[pubmed:30430944|(Abstract)]] |