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Tryptophan
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==Notable studies== * 2010, Sex differences in plasma prolactin response to tryptophan in chronic fatigue syndrome patients with and without comorbid [[fibromyalgia]]<ref>{{Cite journal | last =Weaver | first = Shelley A. | last2 = Janal | first2 = Malvin N. | last3 = Aktan | first3=Nadine | last4 = Ottenweller | first4=John E. | last5 = Natelson | first5 = Benjamin H. | date = May 2010 | title = Sex differences in plasma prolactin response to tryptophan in chronic fatigue syndrome patients with and without comorbid fibromyalgia|url=https://www.ncbi.nlm.nih.gov/pubmed/20384451|journal=Journal of Women's Health (2002)|volume=19|issue=5 | pages = 951β958|doi=10.1089/jwh.2009.1697|issn=1931-843X|pmc=2875960|pmid=20384451|quote= | author-link = Shelley Weaver | authorlink2 = Malvin Janal | author-link3 = Nadine Aktan | authorlink4 = John Ottenweller | authorlink5 = Benjamin Natelson|via=}}</ref> ([https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2875960/ Full text]) * 2011, [[Kynurenine pathway hypothesis|Kynurenine pathway Hypothesis]]: The nature of the chronic Fatigue syndrome (cFs) Revisited<ref>{{Cite journal | last =Blankfield | first=Adele | author-link = Adele Blankfield | date = 2011-07-31 | title = Kynurenine pathway Hypothesis: The nature of the chronic Fatigue syndrome (cFs) Revisited | url =https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3195222/|journal=International Journal of Tryptophan Research : IJTR|volume=4|issue=|pages=47β48|doi=10.4137/IJTR.S7898|issn=1178-6469|pmc=3195222|pmid=22084603|quote=|via=}}</ref> ([https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3195222/ Full text]) * 2012, A Brief Historic Overview of Clinical Disorders Associated with Tryptophan: The Relevance to Chronic Fatigue Syndrome (CFS) and Fibromyalgia (FM)<ref>{{Cite journal | last =Blankfield | first=Adele | author-link = Adele Blankfield | date = 2012-09-17 | title = A Brief Historic Overview of Clinical Disorders Associated with Tryptophan: The Relevance to Chronic Fatigue Syndrome (CFS) and Fibromyalgia (FM)|url=https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3460668/|journal=International Journal of Tryptophan Research : IJTR|volume=5|issue=|pages=27β32|doi=10.4137/IJTR.S10085|issn=1178-6469|pmc=3460668|pmid=23032646|quote=|via=}}</ref> ([https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3460668/ Full text]) * 2014, Tryptophan depletion in chronic fatigue syndrome, a pilot cross-over study<ref>{{Cite journal | last = The | first = Gerard K. H. | last2 = Verkes | first2 = Robbert J. | last3 = Fekkes | first3=Durk | last4 = Bleijenberg | first4 = Gijs| last5 = van der Meer | first5 = Jos W.M. | last6 = Buitelaar | first6 = Jan K. | date = 2014-09-16 | title = Tryptophan depletion in chronic fatigue syndrome, a pilot cross-over study|url=https://www.ncbi.nlm.nih.gov/pubmed/25227994|journal=BMC research notes|volume=7 | pages = 650|doi=10.1186/1756-0500-7-650|issn=1756-0500|pmc=4176591|pmid=25227994|issue=|quote= | author-link = Gerard The | authorlink2 = Robbert Verkes | author-link3 = Durk Fekkes | authorlink4 = Gijs Bleijenberg | authorlink5 = Jos van der Meer|via= | authorlink6 = Jan Buitelaar}}</ref> ([https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4176591/ Full text]) * 2014, Biological phenotypes underpin the physio-somatic symptoms of somatization, depression, and chronic fatigue syndrome<ref>{{Cite journal | last = Anderson | first = G. | last2 = Berk | first2 = M. | last3 = Maes | first3=M. | date = Feb 2014 | title = Biological phenotypes underpin the physio-somatic symptoms of somatization, depression, and chronic fatigue syndrome|url=https://www.ncbi.nlm.nih.gov/pubmed/23952563|journal=Acta Psychiatrica Scandinavica|volume=129|issue=2 | pages = 83β97|doi=10.1111/acps.12182|issn=1600-0447|pmid=23952563}}</ref> ([[pubmed:23952563|Abstract]]) * 2014, Physiosomatic complaints, immune-inflammatory pathways, and serotonin-related mood symptoms: relevance for tryptophan-related challenge procedures and clinical considerations with respect to the DSM-V<ref>{{Cite journal | last =von Polier | first=G. G. | last2 = Zepf | first2 = F. D. | date = Feb 2014 | title = Physiosomatic complaints, immune-inflammatory pathways, and serotonin-related mood symptoms: relevance for tryptophan-related challenge procedures and clinical considerations with respect to the DSM-V|url=https://www.ncbi.nlm.nih.gov/pubmed/24117193|journal=Acta Psychiatrica Scandinavica|volume=129|issue=2 | pages = 98β99|doi=10.1111/acps.12197|issn=1600-0447|pmid=24117193}}</ref> (Comment on 2014, "Biological phenotypes underpin...") * 2016, The Many Neuroprogressive Actions of Tryptophan Catabolites (TRYCATs) that may be Associated with the Pathophysiology of Neuro-Immune Disorders<ref>{{Cite journal | last =Morris | first = Gerwyn | last2 = Carvalho | first2 = AndrΓ© F. | last3 = Anderson | first3=George | last4 = Galecki | first4 = Piotr | last5 = Maes | first5 = Michael | date = 2016 | title=The Many Neuroprogressive Actions of Tryptophan Catabolites (TRYCATs) that may be Associated with the Pathophysiology of Neuro-Immune Disorders|url=https://www.ncbi.nlm.nih.gov/pubmed/26667000|journal=Current Pharmaceutical Design|volume=22|issue=8 | pages = 963β977|issn=1873-4286|pmid=26667000|quote= | author-link = Gerwyn Morris | authorlink2 = AndrΓ© Carvalho | author-link3 = George Anderson | authorlink4 = Piotr Galecki | authorlink5 = Michael Maes|via=}}</ref> ([[pubmed:26667000|Abstract]])
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