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Hypercoagulation hypothesis
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===Mechanism=== One or more [[anaerobic]] viral, bacterial, and/or fungal pathogens activates coagulation pathways. [[Fibrin]] deposits decrease [[oxygen]] in tissues, creating a more hospitable environment for these pathogens.<ref>https://cfsremission.wordpress.com/treatment/thick-blood-clots-dimension-of-cfs-etc/hemex-protocol-and-dave-berg/dave-berg-talk-4/</ref> In his research, Berg found that a large proportion of patients had a tendency to either quickly form fibrin deposits, others to clean up fibrin, while still others had defects in both. Increased blood viscosity results in a chronic lack of oxygen and nutrients to a variety of body systems with effects on the [[muscle]]s, [[brain]], [[HPA axis]] and [[endocrine system]].
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