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Hyperbaric oxygen therapy
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==Notable studies== *2013, The efficacy of hyperbaric oxygen therapy in the management of chronic fatigue syndrome<ref name="Akarsu2013">{{Cite journal | issn = 1066-2936| volume = 40 | issue = 2| pages = 197–200| last1 = Akarsu | first1 = Selim | last2 = Tekin | first2 = Levent | last3 = Ay | first3 = Hakan | last4 = Carli | first4 = Alparslan Bayram | last5 = Tok | first5 = Fatih | last6 = Simşek | first6 = Kemal | last7 = Kiralp | first7 = Mehmet Zeki | title = The efficacy of hyperbaric oxygen therapy in the management of chronic fatigue syndrome| journal = Undersea & Hyperbaric Medicine | date = April 2013 | pmid = 23682549}}</ref> - [https://pubmed.ncbi.nlm.nih.gov/23682549 (Abstract)] ::The [[Fukuda criteria]] was used for CFS patients in this study. *2003, Hyperbaric Therapy in Chronic Fatigue Syndrome<ref name="Hoof2003">{{Cite journal | first = Elke Van | last = Hoof | author-link = Elke Van Hoof | first2 = Danny | last2 = Coomans | first3 = Pascale | last3 = De Bdcker | authorlink3 = Pascale De Becker | first4 = Romain | last4 = Meeusen | first5 = Raymond | last5 = Cluydts | first6 = Kenny | last6 = De Meirleir | authorlink6 = Kenny De Meirleir | date = 2003 | title = Nutritional Supplement (NT Factor™) Restores Mitochondrial Function and Reduces Moderately Severe Fatigue in Aged Subjects|journal=Journal of Chronic Fatigue Syndrome|volume =11|issue=3 | pages = 37-49|doi=10.1300/J092v11n03_04 | url = http://dx.doi.org/10.1300/J092v11n03_04}}</ref> - [https://www.tandfonline.com/doi/abs/10.1300/J092v11n03_04 (Abstract)] <blockquote>Abstract: The aim of this study was to determine if hyperbaric oxygen treatment (HBOT) could be used as adjunctive therapy and if HBOT could increase the quality of life in such a way that the functional status would improve in patients with an infection. A randomized, controlled trial was conducted on 15 ''[[Mycoplasma]]'' sp. infected CFS (CDC 1994) patients and 14 CFS (CDC 1994) patients with no evidence of a ''Mycoplasma'' infection were enrolled in a convenience randomization sample from our referral clinic. No statistical differences were found by use of univariate repeated measures although Bodily Pain as measured by the SF-36 seems to decrease after hyperbaric therapy (Greenhouse-Geisser: p = .010). Trends were found using paired t-testing for ''Mycoplasma'' infected CFS patients. The general perceived fatigue seemed to decrease after hyperbaric therapy (General Fatigue: p = .06). Directly after one week of hyperbaric therapy general fatigue improved (p = .03) but there was a reduction of activity (reduced activity: p = .05) and general perceived health (general health: p = .04). One month later the physical role increased (Role-Physical: p = .07). Although more data is required to make firm conclusions, trends were found. Reduced fatigue, increased levels of activity and an improved reaction time improved significantly their quality of life and therefore, enhanced also their functional status and thus could be used as an adjunctive therapy.<ref name="Hoof2003" /></blockquote> ::Used the [[Fukuda criteria]] * 2018, Prospective Biomarkers from Plasma Metabolomics of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Implicate Redox Imbalance in Disease Symptomatology<ref name="Germain2018">{{Cite journal | date = Dec 2018 | first = Arnaud | last = Germain | authorlink = Arnaud Germain | first2 = David | last2 =Ruppert | authorlink2 = David Ruppert | first3 = Susan M. | last3 = Levine | authorlink3 = Susan Levine | first4 = Maureen R. | last4 = Hanson | authorlink4 = Maureen Hanson | title = Prospective Biomarkers from Plasma Metabolomics of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Implicate Redox Imbalance in Disease Symptomatology|journal=Metabolites |volume=8|issue =4 | pages = 90 | url = https://doi.org/10.3390/metabo8040090|doi=10.3390/metabo8040090}}</ref> - [https://doi.org/10.3390/metabo8040090 (Full text)] <blockquote>Abstract: Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) is a disease of enigmatic origin with no established cure. Its constellation of symptoms has silently ruined the lives of millions of people around the world. A plethora of hypotheses have been vainly investigated over the past few decades, so that the biological basis of this debilitating condition remains a mystery. In this study, we investigate whether there is a disturbance in homeostasis of metabolic networks in the plasma of a female 32-patient cohort compared to 19 healthy female controls. Extensive analysis of the 832-metabolite dataset generated by Metabolon<sup>®</sup>, covering eight biological classes, generated important insight into metabolic disruptions that occur in ME/CFS. We report on 14 metabolites with differences in abundance, allowing us to develop a theory of broad redox imbalance in ME/CFS patients, which is consistent with findings of prior work in the ME/CFS field. Moreover, exploration of enrichment analysis using www.MetaboAnalyst.ca provides information concerning similarities between metabolite disruptions in ME/CFS and those that occur in other diseases, while its biomarker analysis unit yielded prospective plasma biomarkers for ME/CFS. This work contributes key elements to the development of ME/CFS diagnostics, a crucial step required for discovering a therapy for any disease of unknown origin.</blockquote>
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