Skeletal muscle

From MEpedia, a crowd-sourced encyclopedia of ME and CFS science and history

Skeletal muscle are muscles that move bones and other structures in response to voluntary messages from the nervous system. They are one of the three classifications of muscle. The other two categories are cardiac muscle and smooth muscle. Each category of muscle has a different appearance and different role.

Skeletal muscle tissue is composed of long cells called muscle fibers that have a striated appearance and respond the voluntary commands. They connect to bones via connective tissue called tendons. Movement occurs when nerve cells stimulate the muscle to contract.[1]

Studies[edit | edit source]

  • 1992, Skeletal muscle metabolism in the chronic fatigue syndrome. In vivo assessment by 31P nuclear magnetic resonance spectroscopy.[2] (Abstract)
  • 1995, Unusual pattern of mitochondrial DNA deletions in skeletal muscle of an adult human with chronic fatigue syndrome<[3] (First page )
  • 1997, Chronic fatigue syndrome and skeletal muscle mitochondrial function [1]
  • 2013, Cerebral vascular control is associated with skeletal muscle pH in chronic fatigue syndrome patients both at rest and during dynamic stimulation[4] (Full Text)
  • 2015, Abnormalities of AMPK Activation and Glucose Uptake in Cultured Skeletal Muscle Cells from Individuals with Chronic Fatigue Syndrome[5]
  • 2018, Pharmacological activation of AMPK and glucose uptake in cultured human skeletal muscle cells from patients with ME/CFS[6] (Full Text)
  • 2018, Old muscle in young body: an aphorism describing the Chronic Fatigue Syndrome[7] (Full text)

See also[edit | edit source]

Learn more[edit | edit source]

References[edit | edit source]

  1. https://www.visiblebody.com/learn/muscular/muscle-types
  2. Wong, R; Lopaschuk, G; Zhu, G; Walker, D; Catellier, D; Burton, D; Teo, K; Collins-Nakai, R; Montague, T (December 1992). "Skeletal muscle metabolism in the chronic fatigue syndrome. In vivo assessment by 31P nuclear magnetic resonance spectroscopy". Chest. 102 (6): 1716–22. PMID 1446478.
  3. Zhang, C; Baumer, A; Mackay, IR; Linnane, AW; Nagley, P (April 1995). "Unusual pattern of mitochondrial DNA deletions in skeletal muscle of an adult human with chronic fatigue syndrome". Human Molecular Genetics. 4 (4): 751–754. PMID 7633428.
  4. He, Jiabao; Hollingsworth, Kieren G.; Newton, Julia L.; Blamire, Andrew M. (2013). "Cerebral vascular control is associated with skeletal muscle pH in chronic fatigue syndrome patients both at rest and during dynamic stimulation". NeuroImage: Clinical. 2: 168–173. doi:10.1016/j.nicl.2012.12.006.
  5. Brown, Audrey E.; Jones, David E.; Walker, Mark; Newton, Julia L. (2015). "Abnormalities of AMPK Activation and Glucose Uptake in Cultured Skeletal Muscle Cells from Individuals with Chronic Fatigue Syndrome". PLoS One. 10 (4): e0122982. doi:10.1371/journal.pone.0122982. PMC 4383615. PMID 25836975.
  6. Brown, Audrey E.; Dibnah, Beth; Fisher, Emily; Newton, Julia L.; Walker, Mark (2018). "Pharmacological activation of AMPK and glucose uptake in cultured human skeletal muscle cells from patients with ME/CFS". Bioscience Reports. 38 (3): BSR20180242. doi:10.1042/BSR20180242. PMID 29654166.
  7. Pietrangelo, Tiziana; Fulle, Stefania; Coscia, Francesco; Gigliotti, Paola Virginia; Fanò-Illic, Giorgio (September 7, 2018). "Old muscle in young body: an aphorism describing the Chronic Fatigue Syndrome". European Journal of Translational Myology. 28 (3). doi:10.4081/ejtm.2018.7688. ISSN 2037-7460. PMID 30344981.