Nicoletta Carlo-Stella

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Source: LinkedIn

Nicoletta Carlo-Stella, MD, PhD, (b. 1966) is a rheumatologist with the Internal Medicine Department at a hospital in Pavia, Italy.[1][2]

She is one of the authors of the 2011 case definition, International Consensus Criteria.[3]

Studies[edit | edit source]

  • 2011, Excess of activating killer cell immunoglobulin‑like receptors and lack of HLA-Bw4 ligands: a two‑edged weapon in chronic fatigue syndrome.[4]
  • 2010, Letter to the editor re: "A formal analysis of cytokine networks in chronic fatigue syndrome" by Broderick et al.[5]
  • 2009, Demographic and clinical aspects of an Italian patient population with chronic fatigue syndrome.[6]
  • 2009, Molecular study of receptor for advanced glycation endproduct gene promoter and identification of specific HLA haplotypes possibly involved in chronic fatigue syndrome.[7]
  • 2006, A first study of cytokine genomic polymorphisms in CFS: Positive association of TNF-857 and IFNgamma 874 rare alleles.[8]

Talks and interviews[edit | edit source]

Online presence[edit | edit source]

References[edit | edit source]

  1. ↑ https://it.linkedin.com/in/nicoletta-carlo-stella-b0044323
  2. ↑ http://prabook.com/web/person-view.html?profileId=157252
  3. ↑ Carruthers, BM; van de Sande, MI; De Meirleir, KL; Klimas, NG; Broderick, G; Mitchell, T; Staines, D; Powles, A C P; Speight, N; Vallings, R; Bateman, L; Baumgarten-Austrheim, B; Bell, DS; Carlo-Stella, N; Chia, J; Darragh, A; Jo, D; Lewis, D; Light, A; Marshall-Gradisnik, S; Mena, I; Mikovits, JA; Miwa, K; Murovska, M; Pall, ML; Stevens, S (2011), "Myalgic encephalomyelitis: International Consensus Criteria.", Journal of Internal Medicine, 270 (4): 327-38, doi:10.1111/j.1365-2796.2011.02428.x, PMID 21777306
  4. ↑ Pasi, A; Bozzini, S; Carlo-Stella, N; Martinetti, M; Bombardieri, S; De Silvestri, A; Salvaneschi, L; Cuccia, M (2011), "Excess of activating killer cell immunoglobulin‑like receptors and lack of HLA-Bw4 ligands: a two‑edged weapon in chronic fatigue syndrome", Molecular Medicine Reports, 4 (3): 535-40, doi:10.3892/mmr.2011.447
  5. ↑ Carlo-Stella N. (2010) Letter to the editor re: "A formal analysis of cytokine networks in chronic fatigue syndrome" by Broderick et al. Brain, Behavior, and Immunity, 24 (7):1218; author reply 1219. doi: 10.1016/j.bbi.2010.06.004. Epub 2010 Jun 16.Retrieved from http://www.ncbi.nlm.nih.gov/pubmed/20561580
  6. ↑ {{Citation | last1 = Carlo-Stella | first1 = N | authorlink1 = Nicoletta Carlo-Stella | last2 = Cuccia | first2 = M | authorlink2 = | title = Demographic and clinical aspects of an Italian patient population with chronic fatigue syndrome. | journal = Reumatismo | volume = 61 | issue = 4 | page = 285-9 | date = 2009 | pmid = | doi = 10.4081/reumatismo.2009.285
  7. ↑ Carlo-Stella, N; Bozzini, S; De Silvestri, A; Sbarsi, I; Pizzochero, C; Lorusso, L; Martinetti, M; Cuccia, M (2009), "Molecular study of receptor for advanced glycation endproduct gene promoter and identification of specific HLA haplotypes possibly involved in chronic fatigue syndrome", International Journal of Immunopathology and Pharmacology, 22 (3): 745-54, doi:10.1177/039463200902200320, PMID 19822091
  8. ↑ Carlo-Stella, N; Badulli, C; De Silvestri, A; Bazzichi, L; Martinetti, M; Lorusso, L; Bombardieri, S; Salvaneschi, L; Cuccia, M (2006), "A first study of cytokine genomic polymorphisms in CFS: Positive association of TNF-857 and IFNgamma 874 rare alleles", Clinical and Experimental Rheumatology, 24 (2): 179-82, PMID 16762155

antibodies Antibody/immunoglobulin refers to any of a large number of specific proteins produced by B cells that act against an antigen in an immune response.

human leukocyte antigen complex (HLA) - A set of genes responsible for a given person's immune response to potential threats. Specifically, HLA genes encode proteins which help the immune system to distinguish the body's own proteins from proteins which are made by foreign invaders like bacteria and viruses. The HLA complex can vary greatly from person to person, generating unique immune and allergic responses. (Learn more: mecfsresearchreview.me)

cytokine any class of immunoregulatory proteins secreted by cells, especially immune cells. Cytokines are small proteins important in cell signaling that modulate the immune system. (Learn more: me-pedia.org)

myalgic encephalomyelitis (M.E.) - A disease often marked by neurological symptoms, but fatigue is sometimes a symptom as well. Some diagnostic criteria distinguish it from chronic fatigue syndrome, while other diagnostic criteria consider it to be a synonym for chronic fatigue syndrome. A defining characteristic of ME is post-exertional malaise (PEM), or post-exertional neuroimmune exhaustion (PENE), which is a notable exacerbation of symptoms brought on by small exertions. PEM can last for days or weeks. Symptoms can include cognitive impairments, muscle pain (myalgia), trouble remaining upright (orthostatic intolerance), sleep abnormalities, and gastro-intestinal impairments, among others. An estimated 25% of those suffering from ME are housebound or bedbound. The World Health Organization (WHO) classifies ME as a neurological disease.

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