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A Regime for Antiretroviral Treatment of Myalgic Encephalomyelitis
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===Additional phytotherapeutic treatments=== The other pillar of my treatment concept is the additional administration of antiretrovirally active phytotherapeutics. This has enabled us to successfully prevent the use of a synthetic integrase inhibitor such as Raltegravir and also avoid protease inhibitors required in regular anti-[[HIV]] combination therapy. This not only saves a lot of money, but also a range of possible side effects and long-term damage. Furthermore, the risk of resistance development with these phytotherapeutic agents is relatively low. First and foremost [[Cistus Incanus]]. Cistus shows broad antiviral activity in vitro with a low risk of virus resistance. It is in a way effective as an entry inhibitor in [[HIV]] (Rebensburg et al. 2016) and contains several anti-[[HIV]] components. It works at the entry level already, which means that treatment with Cistus blocks the entrance of the virus into the host cell at a very early stage and prevents the docking of virus particles onto cells. This effect provides maximum protection of host cells against virus attack. Many other, synthetic entry inhibitors - according to the study - only take effect at a later stage of the entrance procedure. Cistus, however, already acts as a so-called attachment inhibitor, similar to Pelargonium sidoides, a medicinal plant from South Africa. (Helfer et al. 2014) Cistus is also said to have an advantage over synthetic integrase inhibitors, as it is effective against more [[HIV]] genotypes than, for example, Raltegravir. (Cf. also e.g. Depatureaux et al. 2015) On the side, Cistus also effectively fights upper respiratory tract infections, possesses antibacterial, antiviral and antifungal properties and thus also attacks the so numerous but mostly occult infections present in ME patients. It has also shown in vitro growth-inhibiting activity against Borrelia, with which ME patients are often co-infected. In our case, a total of 2100mg of Cistus in capsule form distributed throughout the day has proven to be an effective dose. It is imperative that Cistus be taken with a time gap of at least two hours between it and [[Viread]]/[[Vemlidy]]. According to our experience, it is probably a good idea to start with Cistus before beginning [[ART]], so that co-infections can be treated in advance. Moreover, this way the organism does not have to get used to two new drugs at the same time. [[Cistus Incanus]] is available in capsules and lozenges, for example, the latter can also be used for immunological reactions such as sore throat or fungal infections in the mouth and throat. With all the intolerances that are so common in ME patients, it is probably better to start treatment with Cistus slowly, too and then gradually increase the dose. [[Reishi]] (Ganoderma lucidum), recommended to us by Dr. Mikovits, strengthens and activates the immune system and has a number of other interesting properties, especially for ME patients, because Reishi also inhibits EBV activation. In addition, according to my literature research, it is effective against [[HIV]]-1 and inhibits [[HIV]]-1 protease. That is why we use it as a protease inhibitor; one capsule (500mg) per day. In addition, one can take [[green tea]] capsules. Some green tea plant extracts have, according to my research, an inhibitory effect on the activity of the Rauscher mouse leukemia virus, which is related to HGRV in some ways, and also on [[HIV]] reverse transcriptase. Green tea contains epigallocatechin- 3 gallate, a powerful antioxidant, which thereby also inhibits the replication of HI viruses and could thus potentially be used as an additional therapy for [[HIV]]-1 infection. According to Dr. Mikovits, in a way it draws retroviruses from the tissue. We take one 250mg green tea capsule daily. Whether our successful, to my knowledge until now unique experiment with a synthetic reverse transcriptase inhibitor in combination with phytotherapeutic entry, protease and reverse transcriptase inhibitors (unique at least with regard to the - high-dose - use of [[Cistus Incanus]] as entry inhibitor and which I consider the most important and effective additional treatment) is applicable to other patients will only become apparent over the course of time. If this does not work for others, chemical integrase inhibitor Raltegravir would most likely have to be added after some time, usually at the latest after one year, namely when progress stagnates or even decline sets in again. Since the research team of the internationally renowned Helmholtz Association of German Research Centres in Munich, which discovered the infection-blocking effect of Cistus against the HI [[retrovirus]], assumes that the antiviral activity of [[Cistus Incanus]] will allow for a dose reduction in anti-[[HIV]] combination therapies and thus to curb side effects and toxicity risk, it does not seem so far-fetched if I postulate the same infection blocking effect of Cistus against HGRV, which may cause the ME disease. The Helmholtz scientists have, by the way, already filed a world patent application.
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